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Project information

ID: GA715322

Fecha inicio

01-03-2017

Fecha fin

28-02-2023


Institutional Coordinator
Universidad Autónoma de Madrid

Financing

1 498 625,00 Euros
(Total amount or amount awarded)

More information in

Analysis of institutional authors

Mittelbrunn Herrero, MariaPrincipal investigatorCarrasco Cerro, ElisaResearcherAranda Gomez, Juan FranciscoResearcherSoto Heredero, GonzaloResearcherOller Pedrosa, JorgeResearcher
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Competitive project

Endolysosomal-mitochondria crosstalk in cell and organism homeostasis

Researchers: Mittelbrunn Herrero, Maria (Investigador principal (IP)); Carrasco Cerro, Elisa (Investigador/a); Aranda Gomez, Juan Francisco (Investigador/a); SOTO HEREDERO, GONZALO (Investigador/a); OLLER PEDROSA, JORGE (Investigador/a)

Affiliations

UAM. Facultad de Ciencias. Biología Molecular (University Department)
UAM. Programa de Doctorado en Biología (Doctorate Program)
UAM. Programa de Doctorado en Biociencias Moleculares (Doctorate Program)
UAM. Carcinogénesis Cutánea (Group)
UAM. Instituto Universitario de Biología Molecular (R & D Center)
UAM. Facultad de Ciencias. Biología (University Department)
UAM. Universidad Autónoma de Madrid (University)
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Abstract

Control of Inflammation by Calorie Restriction Mimetics: On the Crossroad of Autophagy and Mitochondria (Most relevant results); Control of Inflammation by Calorie Restriction Mimetics: On the Crossroad of Autophagy and Mitochondria (Summary)
Keywords
Age associated-diseasesCardiorenal syndromeCell metabolismEndolysosomal systemImmunologyInflammagingInterorganelle communicationMetabolic disorderMitochondriaT lymphocytes

Financing

Quality index

Call

EXCELLENT SCIENCE - European Research Council (ERC); ERC Starting Grant

Financer

H2020

Reach

Unión Europea

Institutional Coordinator

Si

Related items

Type of RelationshipReferenceExternal Code/IDLink
Output ofControl of Inflammation by Calorie Restriction Mimetics: On the Crossroad of Autophagy and Mitochondria10.3390/cells9010082View on iMarina
Output ofMitochondrial dysfunction defines T cell exhaustion10.1016/j.cmet.2021.02.010View on iMarina
Output ofGlycolysis – a key player in the inflammatory response10.1111/febs.15327View on iMarina
Output ofExtremely Differentiated T Cell Subsets Contribute to Tissue Deterioration During Aging10.1146/annurev-immunol-101721-064501View on iMarina
Output ofT cells with dysfunctional mitochondria induce multimorbidity and premature senescence10.1126/science.aax0860View on iMarina
Output ofNicotinamide adenine dinucleotide metabolism in the immune response, autoimmunity and inflammageing10.1111/bph.15477View on iMarina
Output ofMitochondrial activity in T cells10.1016/j.mito.2017.10.006View on iMarina
Output ofInterorganelle Communication between Mitochondria and the Endolysosomal System10.3389/fcell.2017.00095View on iMarina
Output ofRewiring Vascular Metabolism Prevents Sudden Death due to Aortic Ruptures-Brief Report10.1161/ATVBAHA.121.317346View on iMarina
Output ofThe role of T cells in age-related diseases10.1038/s41577-021-00557-4View on iMarina
Output ofHallmarks of T cell aging10.1038/s41590-021-00927-zView on iMarina
Output ofExtracellular Tuning of Mitochondrial Respiration Leads to Aortic Aneurysm10.1161/CIRCULATIONAHA.120.051171View on iMarina
Output ofPeriodic Reporting for period 4 - EndoMitTalk (Endolysosomal-mitochondria crosstalk in cell and organism homeostasis)