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Analysis of institutional authors

Pascual-Castroviejo ICorresponding AuthorPascual-Pascual SAuthorCarceller-Benito FCorresponding Author

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Estenosis del acueducto de Silvio en la neurofibromatosis tipo 1: Presentación de 19 pacientes infantiles

Publicated to:REVISTA DE NEUROLOGIA. 45 (1): 18-21 - 2007-01-01 45(1), DOI: 10.33588/rn.4501.2006344

Authors: Pascual-Castroviejo I; Pascual-Pascual S; Velázquez-Fragua R; Viaño J; Carceller-Benito F

Affiliations

Hospital Universitario La Paz - Author
Sanatorio de Nuestra Señora del Rosario - Author

Abstract

To present a series of infantile patients with aqueductal stenosis associated with neurofibromatosis type 1 (NF1).Nineteen patients with ages below 16 years, 11 girls and 8 boys, with NF1 presented hydrocephalus due to aqueductal stenosis. All patients, except one who died before the imaging study was performed and was diagnosed by autopsy, were studied by pneumoencephalography (since 1965 to 1974), computerized tomography (CT) (since 1975 to 1984), magnetic resonance (MR) or MR and CT (since 1985 to 2004) (two children had been studied by pneumoencephalography some years before) most times to discard optic pathway tumor and, in few patients, because of intracranial hypertension.All patients showed three ventricular hydrocephalus with aqueductal stenosis. Eleven patients showed optic pathway tumor. One patient had a benign aqueductal tumor that impaired the normal flow of cerebrospinal fluid. Neurological features of hydrocephalus occurred very rapidly in some patients and after several years of evolution in others. Two boys showed precocious puberty. All patients were treated with shunt.In our series, aqueductal stenosis occurred in about 5% of children with NF1. Aqueductal stenosis and hydrocephalus were identified at a short age because many patients were studied suspecting optic pathway tumor. Eleven patients (about 60%) associated optic pathway tumor and aqueductal stenosis.

Keywords

Aqueductal stenosisAqueductal tumorHydrocephalusNeurofibromatosis type 1 (nf1)Optic pathway tumor

Quality index

Bibliometric impact. Analysis of the contribution and dissemination channel

The work has been published in the journal REVISTA DE NEUROLOGIA, and although the journal is classified in the quartile Q4 (Agencia WoS (JCR)), its regional focus and specialization in Clinical Neurology, give it significant recognition in a specific niche of scientific knowledge at an international level.

From a relative perspective, and based on the normalized impact indicator calculated from the Field Citation Ratio (FCR) of the Dimensions source, it yields a value of: 2.32, which indicates that, compared to works in the same discipline and in the same year of publication, it ranks as a work cited above average. (source consulted: Dimensions Jun 2025)

Specifically, and according to different indexing agencies, this work has accumulated citations as of 2025-06-28, the following number of citations:

  • WoS: 2
  • Scopus: 15
  • Europe PMC: 2

Impact and social visibility

From the perspective of influence or social adoption, and based on metrics associated with mentions and interactions provided by agencies specializing in calculating the so-called "Alternative or Social Metrics," we can highlight as of 2025-06-28:

  • The use of this contribution in bookmarks, code forks, additions to favorite lists for recurrent reading, as well as general views, indicates that someone is using the publication as a basis for their current work. This may be a notable indicator of future more formal and academic citations. This claim is supported by the result of the "Capture" indicator, which yields a total of: 10 (PlumX).

Leadership analysis of institutional authors

There is a significant leadership presence as some of the institution’s authors appear as the first or last signer, detailed as follows: First Author (PASCUAL CASTROVIEJO, IGNACIO) and Last Author (CARCELLER BENITO, FERNANDO).

the authors responsible for correspondence tasks have been PASCUAL CASTROVIEJO, IGNACIO and CARCELLER BENITO, FERNANDO.